Y. Murakami and S. Ishimitsu, “Generation Mechanisms of Two-Tone Suppression Using a Cochlear Model,” in Proc. AES Conference: 45th International Conference: Applications of Time-Frequency Processing in Audio, Mar. 2012, Paper 6-5. [Online]. Available: https://aes.org/publications/elibrary-page/?id=16172
Murakami Y, Ishimitsu S. Generation Mechanisms of Two-Tone Suppression Using a Cochlear Model. In: AES Conference: 45th International Conference: Applications of Time-Frequency Processing in Audio. Audio Engineering Society; 2012. Paper 6-5. Available from: https://aes.org/publications/elibrary-page/?id=16172
@inproceedings{Murakami2012_16172,
author = {Murakami, Yasuki and Ishimitsu, Shunsuke},
title = {{Generation Mechanisms of Two-Tone Suppression Using a Cochlear Model}},
booktitle = {AES Conference: 45th International Conference: Applications of Time-Frequency Processing in Audio},
note = {Paper 6-5},
year = {2012},
month = mar,
publisher = {Audio Engineering Society},
url = {https://aes.org/publications/elibrary-page/?id=16172}
}
TY - CPAPER
TI - Generation Mechanisms of Two-Tone Suppression Using a Cochlear Model
AU - Murakami, Yasuki
AU - Ishimitsu, Shunsuke
T2 - AES Conference: 45th International Conference: Applications of Time-Frequency Processing in Audio
M1 - Paper 6-5
PY - 2012
DA - 2012/03/06
UR - https://aes.org/publications/elibrary-page/?id=16172
PB - Audio Engineering Society
LA - en
AB - A cochlear model including a nonlinear function for somatic motility of the outer hair cell (OHC) has been proposed for compression relating to cochlear nonlinearity evolved by a tone. However, the mechanisms of two-tone suppression, also known as cochlear nonlinearity, are not yet clear. The present study investigates the mechanisms of two-tone suppression in a simulation using the previously proposed cochlear model. Twotone suppression by an OHC model is thus presented. The simulation results are consistent with the results of physiological experiments and show the possibility that two-tone suppressiona is generated by the somatic motility of the OHC.
ER -